MOTS c is an exercise induced mitochondrial encoded regulator of age dependent physical decline and muscle homeostasis Nature Communications MOTS c improves intrinsic muscle mitochondrial bioenergetic health and efficiency in a PGC 1 AMPK dependent manner ScienceDirect Frontiers MOTS c repairs myocardial damage by inhibiting the CCN1 ERK1 2 EGR1 pathway in diabetic rats MOTS c reduces myostatin and muscle atrophy signaling American Journal of Physiology Endocrinology and Metabolism American Physiological Society Peptide Therapy for Anti Aging, Immunity & Wellness Miami Stem Cell
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